<span style="font-family: arial, prescription helvetica,sans-serif;”>How does low-level-laser-treatment affect fat cells?
I will begin to walk you through a series of primary and secondary reactions that originate in the mitochondria of a single cell and can migrate throughout the whole body!
The mitochondrion is an energy station for all eukaryotic cells, and that energy produced in the mitochondrion is what provides life to the entire organism. The mitochondrion is where adenosine tri-phosphate (ATP) is produced, an essential molecule driving many reactions.
The mitochondrion is the specific target for Erchonia’s low-level-laser devices. Specifically, cytochrome c oxidase, a terminal enzyme, is targeted by the low-level-laser. Cytochrome c oxidase is a photoacceptor, absorbing light at a peak spectrum of 630-670nm (red spectrum). This particular molecule is responsible for ensuring that the Respiratory Chain goes to completion. The Respiratory Chain harvests electrons from O2 and NADH passing them along through a series of Redox reactions, ultimately producing ATP and H2O. Cytochrome c oxidase promotes the electron flow along the Respiratory Chain between Complexes III and IV.
Low-level-laser is proposed, based on a study performed in 2005, to stimulate photoexcitation of certain reaction centers in the cytochrome c oxidase molecule (like CuA and CuB) influencing the redox state of these molecules, and consequently, the rate of the electron flow in the molecule; meaning, photoexcitation of cytochrome c oxidase may lead to redox changes and modulations of biochemical reactions through a cascade of reactions called photosignal transduction (stimulation of other reactions).